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Vitamin B6 (pyridoxine) DNA health report

Follow the link of the selected polymorphism to read a brief description of how the selected polymorphism affects Vitamin B6 (pyridoxine) and see a list of existing studies.

SNP polymorphisms related to the topic Vitamin B6 (pyridoxine):

rs885813Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs1106797The gene encodes pyridoxal kinase, which converts inactive vitamin B6 into the active cofactor p-5-p. Breakage can cause polyneuropathy, and high doses of p-5-p are recommended.
rs1256335Breakage causes a deficiency of the active form of vitamin B6, pyridoxal-5'-phosphate (p-5-p). Supplemental intake of p-5-p is recommended.
rs1256348Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs1772719Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs2236225Possible increased risk of fetal birth defects. A 1.5-fold higher risk for Caucasian mothers to give birth to children with DNT (neural tube defect). The association in children with this mutation with an increased risk of heart defects is greater if their mother did not get enough folic acid during pregnancy. The risk is reduced with adequate levels of folic acid and vitamin B6.
rs2242420Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs2275370Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs2276528The gene encodes pyridoxal kinase, which converts inactive vitamin B6 into the active cofactor p-5-p. Breakage can cause polyneuropathy, and high doses of p-5-p are recommended.
rs3767150Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs3767155Risk of a 1.4-fold decrease in the active form of vitamin B6 in blood and cerebrospinal fluid.
rs4654748Breakage causes a deficiency of the active form of vitamin B6, pyridoxal-5'-phosphate (p-5-p). Supplemental intake of p-5-p is recommended.
rs5742905Responsible for vitamin B6 susceptibility in homocystinuria.
rs8128639The gene encodes pyridoxal kinase, which converts inactive vitamin B6 into the active cofactor p-5-p. Breakage can cause polyneuropathy, and high doses of p-5-p are recommended.
rs11079804
rs13050307The gene encodes pyridoxal kinase, which converts inactive vitamin B6 into the active cofactor p-5-p. Breakage can cause polyneuropathy, and high doses of p-5-p are recommended.
rs17679445Responsible for reducing the activity of the enzyme pyridoxamine-5-phosphatoxidase, leads to a significant decrease in the level of the active form of vitamin B6.
rs121964972Breakage increases the likelihood of homocystinuria, which is insensitive to vitamin B6.
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