Zinc finger protein
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Zinc finger proteins constitute the most extensive transcription factor family within the human genome. Their diverse configurations and functions make them adaptable to various biological processes, encompassing development, differentiation, metabolism, and autophagy. Recent decades have witnessed growing evidence implicating zinc finger proteins in cancer advancement. Nevertheless, the mechanisms underlying their involvement in cancer progression differ among various cancer types and can even vary within the same cancer type under different stress conditions. In this discussion, we explore the general mechanisms of zinc finger proteins in transcription regulation and provide an overview of recent research concerning their roles in cancer progression.
Follow the link of the selected polymorphism to read a brief description of how the selected polymorphism affects Zinc and see a list of existing studies.
SNP polymorphisms related to the topic Zinc:
| rs13266634 | The zinc transporter gene SLC30A8 polymorphism is associated with type 2 diabetes. |
| rs1805087 | Moderate risk factor for hyperhomocysteinaemia. A factor that increases the body's need for folic acid and vitamin B12. |
| rs11558471 | In this breakdown, zinc intake can cause an increase in fasting glucose levels. |
| rs2769264 | Gene variants affecting copper, selenium and zinc content in blood. |
| rs10925257 | A marker of vitamin B12 deficiency and frailty in older women. |
| rs2466293 | A gene for autoantibodies to a zinc transporter, is associated with an increased risk of developing diabetes mellitus. |
| rs4908107 | |
| rs1505521 | |
| rs4646437 | |
| rs2072704 | |
| rs55901263 | |
| rs111811483 | |
| rs11781136 | |
| rs2047962 | |
| rs12522805 | |
| rs527392 | |
| rs1568569 | |
| rs595641 | |
| rs17366568 | |
| rs7113940 | |
| rs3821799 | |
| rs185949718 | |
| rs7678298 | |
| rs17060812 | |
| rs896378 | |
| rs10488695 | |
| rs13427170 | |
| rs7833266 | |
| rs182052 | |
| rs883396 | |
| rs2241767 | |
| rs1926740 | |
| rs11818989 | |
| rs692570 | |
| rs2497766 | |
| rs12358488 | |
| rs2497756 | |
| rs6482124 | |
| rs2497760 | |
| rs7893618 | |
| rs573264 | |
| rs402311 | |
| rs575707 | |
| rs848189 | |
| rs3781998 | |
| rs530532 | |
| rs1050631 | |
About The Author
Li Dali
Li Dali, a National Foundation for Outstanding Youth Fund recipient, is a researcher at the School of Life Sciences in East China Normal University. He earned his PhD in genetics from Hunan Normal University in 2007 and conducted collaborative research at Texas A&M University during his doctoral studies. Li Dali and his team have optimized and innovated gene editing technology, leading to the establishment of a world-class system for constructing gene editing disease models. Wilson's disease is an autosomal recessive condition, necessitating that both parents carry at... While the body requires only minuscule quantities of the trace mineral molybdenum, it serves as a... Hereditary hemochromatosis is a genetic condition associated with severe liver disease and various...
